Genetic heterogeneity of motor neuropathies

نویسندگان

  • Boglarka Bansagi
  • Helen Griffin
  • Roger G. Whittaker
  • Thalia Antoniadi
  • Teresinha Evangelista
  • James Miller
  • Mark Greenslade
  • Natalie Forester
  • Jennifer Duff
  • Anna Bradshaw
  • Stephanie Kleinle
  • Veronika Boczonadi
  • Hannah Steele
  • Venkateswaran Ramesh
  • Edit Franko
  • Angela Pyle
  • Hanns Lochmüller
  • Patrick F. Chinnery
  • Rita Horvath
چکیده

OBJECTIVE To study the prevalence, molecular cause, and clinical presentation of hereditary motor neuropathies in a large cohort of patients from the North of England. METHODS Detailed neurologic and electrophysiologic assessments and next-generation panel testing or whole exome sequencing were performed in 105 patients with clinical symptoms of distal hereditary motor neuropathy (dHMN, 64 patients), axonal motor neuropathy (motor Charcot-Marie-Tooth disease [CMT2], 16 patients), or complex neurologic disease predominantly affecting the motor nerves (hereditary motor neuropathy plus, 25 patients). RESULTS The prevalence of dHMN is 2.14 affected individuals per 100,000 inhabitants (95% confidence interval 1.62-2.66) in the North of England. Causative mutations were identified in 26 out of 73 index patients (35.6%). The diagnostic rate in the dHMN subgroup was 32.5%, which is higher than previously reported (20%). We detected a significant defect of neuromuscular transmission in 7 cases and identified potentially causative mutations in 4 patients with multifocal demyelinating motor neuropathy. CONCLUSIONS Many of the genes were shared between dHMN and motor CMT2, indicating identical disease mechanisms; therefore, we suggest changing the classification and including dHMN also as a subcategory of Charcot-Marie-Tooth disease. Abnormal neuromuscular transmission in some genetic forms provides a treatable target to develop therapies.

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عنوان ژورنال:

دوره 88  شماره 

صفحات  -

تاریخ انتشار 2017